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With the introduction of the natural testosterone boosting matrix,Restore, AlR Industries has once again placed itself firmly ahead of the competition. With Restore,on sale now at Paramount-Supplements.com, you can expect a potent testosterone booster with the added benefit of lower estrogen levels,lower cortisol levels, and a boosted sex drive. Restore is a male optimization formula designed to exceed your muscle building anabolic expectation. Let't take a look at what makes Restore from ALRI such an amazing supplement.

Time to Restore™

ALR Industries Restore™ is not just another testosterone booster. Though an increase in testosterone production alone is pretty cool, and one of the reported major benefits of Restore™, it would be only part of the Restore™ optimized male matrix. Instead we have opted to see how many arguments we can start (and knock-offs we can inspire) this time by raising the bar for product expectations beyond what any one product can currently offer performance oriented males…by doing the whole job. Its okay, we are men, we like competition!

Estrogen Control

6-Bromodione: There are two types of estrogen aromatase inhibitors and application of only one leaves the other pathway open for feminizing potential.

6 alpha-Bromodione is a competitive inhibitor of aromatase enzymes. Basically this means it works by way of binding to the active binding site of the aromatase enzyme resulting in prevention of interaction with other steroids that aromatize like testosterone and the rest of our favorite male androgens. This is great for rapid binding and short term aromatase control, but unfortunately a competitive inhibitor will eventually let go of the enzyme and allow it to do feminizing things to manly hormones. So, only half of the job is done, but it has its important role as well. 6 beta-Bromodione is a mechanism-based irreversible inhibitor of the aromatase enzyme. An irreversible aromatase inhibitor is also referred to as a suicide inhibitor. It’s pretty cool in that it acts similar to a competitive inhibitor in the way it binds, but like a pissed-off ex-girlfriend, it is both highly selective and it will not let go until death do they part. Together the two analogs in 6-Bromodione make sure that estrogen is under control, and many would assume that less estrogen means the prolactin would be under control as well. Often, once prolactin increases are stimulated, shutting it back down takes some extra effort and a protracted period of time. How long are you willing to wait to be an optimized male?

Prolactin Inhibition

Many have heard of or used drugs like bromocriptine that inhibit prolactin production for medical needs. Of course one of the benefits is an often reported increase in libido, and “related factors”, but the side-effects such as nausea and dry mouth can certainly negate the positive.

Prolact-X™ (Chasteberry Diterpine Matrix Extract) There has been a long history of supplemental use of Chasteberry for treatment of PMS in women. Most researches believe that PMS is commonly the result of high prolactin levels so no surprise that we took an interest in this herb.

The amino acid dopamine is the body’s natural controller of prolactin release. Of course many dopaminergic compounds have been noted as effective treatments for increasing GH secretion as well, which correlates rather well with optimization in male performance due to the fact that elevated prolactin results in decreased GH and IGF-1 production.

The search for the prolactin-suppressive principles in Chasteberry provided a number of compounds with dopaminergic properties. In short, research showed that they bound to recombinant dopamine DA2-receptors and suppressed prolactin release from cultivated lactotrophs as well as in animal experiments. The search for the chemical identity of the dopaminergic compounds resulted in isolation of a number of diterpenes of which some clerodadienols were most important for the prolactin-suppressive effects. They were almost more effective in their prolactin-suppressive properties than dopamine itself.

Naturally just crushing up a bunch of Chasteberry is not likely to do the job, but proper preparation of the de-feminizing herb has finally given us an effective synergist to support male optimization.

So, cool, now that we have dealt with the HPTA suppression and feminizing issues. By doing so the obvious result is a notable and welcomed increase in testosterone production. But hey, why not push all the way and reach for optimization?

Increased Free Testosterone

Free-T™ (Proprietary Pure Avenacosides Matrix) Seems most males who use supplements for either lean mass gains, libido augmentation or both have heard of Avena Sativa. Yeah, I know, lots of products already have some form avena sativa or its extracts in it, so what makes this one special? Of course we used an extraction process for maximum active compound content (greatest possible amount of active milligrams per capsule), but in truth Restore™ as a product is special, our Free-T™ (Proprietary Pure Avenacosides Matrix) from avena sativa just works well in the whole matrix to provide maximum results.

In the body testosterone travels around in two forms: Bound or inactive…meaning it is bound to a protein called SHBGB, and Unbound or free…meaning it is free to do manly things like build muscle and increase your capacity to perform sexually.

Basically there are phytochemicals in avena sativa called avenacosides A and B. Some studies indicate avenacosides increase actual active or free testosterone in the body by freeing up the bound testosterone from SHBG so there is more active testosterone. What good is it to have all kinds of extra testosterone if it is playing S & M games with SHBG?

Cortisol Inhibition

MbAET Unless you went coma bound in the early 90’s you have been aware of the on going benefits reaped and progress made with DHEA and its even better metabolites. As an example are the patented and effective products 7-OXO-DHEA and of course 7-Hydroxy-DHEA analogs. Since these analogs prevent conversion into androgenic metabolites they have been touted by many as the best thing since pizza. Considering the lack of androgenic side effects possible while promoting fat loss, lean mass retention and even maximizing thyroid gland activity, not a real surprise they have become very popular. Of course oral bioavalability is pretty poor with most of these analogs thus requiring higher dosages.

Likely the most powerful and effective DHEA analog is b-AET (beta-androstenetriol). It has been shown in studies to be between 100 and 100,000 times more active than its DHEA precursor metabolites. However, like most DHEA analogs there is the issue of poor oral bioavailability. By supporting the delivery value through simple alkylation, MbAET is nearly 100% orally bioavailable and only takes a few milligrams to do its job.

Yeah. I know, get to the fat loss and lean muscle side of things in regard to MbAET…

Glucocorticoids in humans are in two forms. Inactive cortisone and very active in eating muscle cortisol. There are two enzymes that are able to make each of these convert into the other.

11b-HSD-1: Converts inactive cortisone into cannibalistic cortisol. Studies have implicated this event in fat tissue as a pathway for increased fat storage. Part of the reason GH has a positive affect upon body composition is through its ability to inhibit 11b-HDS-1.

11b-HSD-2: Converts nasty cortisol into cortisone. 11betaHSD2 debulks intracellular cortisol by 90%. (Let the 11b-HSD-2 rule the house)

Hmmm, so, more 11b-HSD-1 means more cortisol which eats more muscle that likes to help fat do ugly things.

And less 11b-HSD-1 means…

MbAET inhibits the 11b-HSD-1 enzyme both locally and systemically. This means that there is less conversion of cortisone to cortisol. Based upon the studies it appears that in mediating this pathway, it increases immune function and recovery of cells as well. Less cortisol and fat, more lean muscle and positive support to health. Not bad!

Optimized Delivery

CYP-X™ Proprietary Grapefruit Extract)

There were several studies some years back that showed a dramatic increase in absorption for some compounds when taken with grapefruit juice. The reason was due to a specific part of grapefruit called dihydoxybergamottin (DHB). It decreases the gastronomical destruction of compounds susceptible to the CYP-450 enzyme thus allowing a far greater amount of the compound to pass into the body’s circulatory system instead of the toilet. The components of Restore™ are a good example…

By creating an ultra pure active extract of dihydoxybergamottin (DHB) called CYP-X™ we have maximally increased the overall oral bioavailability of our synergistic compounds in Restore™ to allow both results and cost effective value in one product.

Don’t Settle…

Having anything less than optimal testosterone levels is unacceptable for any male wanting to be the best they can be in all things male, but as a man excess estrogen and cortisol is a bitch. Restore™ from ALR Industries is the newest complete synergistic weapon in the fight against mediocrity for all men.

Restore™

Suggested Retail Price $69.99 Paramount-Supplements Price: $29.99

Restore™ is a great product for anyone wanting to be an optimized male. Restore™ also stacks extremely well with Jungle Warfare.



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Research References:

1) Environment, human reproduction, menopause, and andropause. Environ Health Perspect. 1993 Jul;101 Suppl 2:91-100. Review.

2) Acute exposure of adult male rats to dietary phytoestrogens reduces fecundity and alters epididymal steroid hormone receptor expression. J Endocrinol. 2006 Jun;189(3):565-73.

3) [Phytoestrogens and soy foods in infants and children: caution is needed] Arch Pediatr. 2006 Mar;13(3):235-7. Epub 2006 Feb 3.

4) Genistein, a phytoestrogen, effectively modulates luteinizing hormone and prolactin secretion in ovariectomized ewes during seasonal anestrus. Neuroendocrinology. 2004 Feb;79(2):73-81.

5) Antiestrogens are partial estrogen agonists for prolactin production in primary pituitary cultures. Mol Cell Endocrinol. 1986 Dec;48(2-3):127-33.

6) Stereochemistry of the functional group determines the mechanism of aromatase inhibition by 6-bromoandrostenedione. Endocrinology. 1987 Sep;121(3):1010-6.

7) Chaste tree (Vitex agnus-castus)--pharmacology and clinical indications.

8) Phytomedicine. 2003 May;10(4):348-57. Review.Immunopharmacol Immunotoxicol. 2005;27(1):15-32. Molecular specificity of 5-androstenediol as a systemic radioprotectant in mice. Whitnall MH, Villa V, Seed TM, Benjack J, Miner V, Lewbart ML, Dowding CA, Jackson WE 3rd.

9) J Clin Endocrinol Metab. 2005 Apr;90(4):2015-21. Epub 2005 Jan 25. Urinary markers of adrenarche: reference values in healthy subjects, aged 3-18 years. Remer T, Boye KR, Hartmann MF, Wudy SA. Department of Nutrition and Health, Research Institute of Child Nutrition, Heinstuck 11, 44225 Dortmund, Germany. remer@fke-do.de

10) Rinsho Byori. 1998 Jun;46(6):505-17. Control of the immune response by DHEA and its metabolites. Loria RM, Padgett DA. Department of Microbiology, Virginia Commonwealth University, Medical College of Virginia, Richmond 23298-09678, USA.

11) Ann N Y Acad Sci. 2000;917:860-7. Androstenetriol and androstenediol. Protection against lethal radiation and restoration of immunity after radiation injury. Loria RM, Conrad DH, Huff T, Carter H, Ben-Nathan D.

12) J Endocrinol. 2000 Feb;164(2):161-9. Conversion of dehydroepiandrosterone to downstream steroid hormones in macrophages. Schmidt M, Kreutz M, Loffler G, Scholmerich J, Straub RH. Institute of Biochemistry, Genetics and Microbiology, University of Regensburg, Germany.

13) J Neuroimmunol. 1998 Apr 1;84(1):61-8. Endocrine regulation of murine macrophage function: effects of dehydroepiandrosterone, androstenediol, and androstenetriol. Padgett DA, Loria RM. Department of Oral Biology, College of Dentistry, The Ohio State University, Columbus 43210, USA. padgett.11@osu.edu

14) Psychoneuroendocrinology. 1997;22 Suppl 1:S103-8. Antiglucocorticoid function of androstenetriol. Loria RM. Virginia Commonwealth University, Medical College of Virginia, Richmond 23298-0678, USA. Loria@gems.vcu.edu

15) Ann N Y Acad Sci. 1992 Apr 15;650:363-6. Mobilization of cutaneous immunity for systemic protection against infections. Loria RM, Padgett DA. Virginia Commonwealth University, School of Basic Health Sciences, Medical College of Virginia, Richmond 23298.

16) Obes Res. 2005 Jul;13(7):1157-66. Increased cortisol bioavailability, abdominal obesity, and the metabolic syndrome in obese women. Duclos M, Marquez Pereira P, Barat P, Gatta B, Roger P

17) Laboratoire Neurogenetique et Stress, INSERM U471, Institut Francois Magendie, Universite Bordeaux II, rue C. Saint Saens, 33077 Bordeaux Cedex, France. duclos@pop.bordeaux.inserm.fr.

18) Horm Metab Res. 2005 Apr;37(4):193-7. Obesity and cortisol status. Salehi M, Ferenczi A, Zumoff B. Division of Endocrinology and Metabolism, Department of Medicine, Beth Israel Medical Center and Albert Einstein College of Medicine, New York, NY 10003, USA.

19) Int J Parasitol. 2004 Nov;34(12):1405-12. Effect of artemether alone and in combination with grapefruit juice on hepatic drug-metabolising enzymes and biochemical aspects in experimental Schistosoma mansoni.

20) Inhibition of in-vitro simvastatin metabolism in rat liver microsomes by bergamottin, a component of grapefruit juice. J Pharm Pharmacol. 2004 Aug;56(8):1007-14.

WARNING: NOT FOR USE BY INDIVIDUALS UNDER THE AGE OF 21 YEARS. DO NOT USE IF PREGNANT OR NURSING. KEEP OUT OF REACH OF CHILDREN. Do NOT consume this product if you have a medical condition and/or taking any prescription medication. Do not exceed recommended serving. Discontinue use and call a physician or licensed qualified health care professional immediately if you experience rapid heartbeat, dizziness, severe headache, or other similar symptoms.

These statements have not been evaluated by the FDA. This product is not intended to diagnose, treat, or prevent any disease.